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MSH2

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出典 ^ a b c ⇒GRCh38: Ensembl release 89: ENSG00000095002 - Ensembl, May 2017
^ a b c ⇒GRCm38: Ensembl release 89: ENSMUSG00000024151 - Ensembl, May 2017
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^ Mouse PubMed Reference:
^ a b “Transcription-coupled repair deficiency and mutations in human mismatch repair genes”. Science 272 (5261): 557?60. (April 1996). Bibcode: 1996Sci...272..557M. doi:10.1126/science.272.5261.557. PMID 8614807. https://zenodo.org/record/1231074.
^ a b “Inactivation of the mouse Msh2 gene results in mismatch repair deficiency, methylation tolerance, hyperrecombination, and predisposition to cancer”. Cell 82 (2): 321?30. (July 1995). doi:10.1016/0092-8674(95)90319-4. PMID 7628020.
^ a b “Reduced host cell reactivation of oxidative DNA damage in human cells deficient in the mismatch repair gene hMSH2”. Mutagenesis 22 (3): 235?43. (May 2007). doi:10.1093/mutage/gem008. PMID 17351251.
^ “Refinement of evolutionary medicine predictions based on clinical evidence for the manifestations of Mendelian diseases”. Scientific Reports 9 (1): 18577. (December 2019). Bibcode: 2019NatSR...918577S. doi:10.1038/s41598-019-54976-4. PMC 6901466. PMID 31819097. https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6901466/.
^ “Mismatch repair and the hereditary non-polyposis colorectal cancer syndrome (HNPCC)”. Cancer Invest. 20 (1): 102?9. (2002). doi:10.1081/cnv-120000371. PMID 11852992.
^ “Structure of the human MutSalpha DNA lesion recognition complex”. Mol. Cell 26 (4): 579?92. (May 2007). doi:10.1016/j.molcel.2007.04.018. PMID 17531815.
^ “Hereditary nonpolyposis colorectal cancer: diagnostic strategies and their implications”. Evid Rep Technol Assess (Full Rep) (150): 1?180. (May 2007). PMC 4781224. PMID 17764220. https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4781224/.
^ “Era of universal testing of microsatellite instability in colorectal cancer”. World J Gastrointest Oncol 5 (2): 12?9. (February 2013). doi:10.4251/wjgo.v5.i2.12. PMC 3613766. PMID 23556052. https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3613766/.
^ “Interaction between the Msh2 and Msh6 nucleotide-binding sites in the Saccharomyces cerevisiae Msh2-Msh6 complex”. J. Biol. Chem. 285 (12): 9301?10. (March 2010). doi:10.1074/jbc.M109.096388. PMC 2838348. PMID 20089866. https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2838348/.
^ “Isolation of an hMSH2-p160 heterodimer that restores DNA mismatch repair to tumor cells”. Science 268 (5219): 1909?12. (June 1995). Bibcode: 1995Sci...268.1909D. doi:10.1126/science.7604264. PMID 7604264.
^ “Nuclear translocation of mismatch repair proteins MSH2 and MSH6 as a response of cells to alkylating agents”. J. Biol. Chem. 275 (46): 36256?62. (November 2000). doi:10.1074/jbc.M005377200. PMID 10954713.
^ “Structural, molecular and cellular functions of MSH2 and MSH6 during DNA mismatch repair, damage signaling and other noncanonical activities”. Mutat. Res. 743?744: 53?66. (February 2013). doi:10.1016/j.mrfmmm.2012.12.008. PMC 3659183. PMID 23391514. https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3659183/.
^ a b c “Interactions of human hMSH2 with hMSH3 and hMSH2 with hMSH6: examination of mutations found in hereditary nonpolyposis colorectal cancer”. Mol. Cell. Biol. 18 (11): 6616?23. (November 1998). doi:10.1128/mcb.18.11.6616. PMC 109246. PMID 9774676. https://www.ncbi.nlm.nih.gov/pmc/articles/PMC109246/.
^ “Large conformational changes in MutS during DNA scanning, mismatch recognition and repair signalling”. EMBO J. 31 (11): 2528?40. (May 2012). doi:10.1038/emboj.2012.95. PMC 3365432. PMID 22505031. https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3365432/.
^ “Functional studies and homology modeling of Msh2-Msh3 predict that mispair recognition involves DNA bending and strand separation”. Mol. Cell. Biol. 30 (13): 3321?8. (July 2010). doi:10.1128/MCB.01558-09. PMC 2897569. PMID 20421420. https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2897569/.
^ “Mechanism of mismatch recognition revealed by human MutSβ bound to unpaired DNA loops”. Nat. Struct. Mol. Biol. 19 (1): 72?8. (January 2012). doi:10.1038/nsmb.2175. PMC 3252464. PMID 22179786. https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3252464/.
^ a b c “MSH2 and ATR form a signaling module and regulate two branches of the damage response to DNA methylation”. Proc. Natl. Acad. Sci. U.S.A. 100 (26): 15387?92. (December 2003). Bibcode: 2003PNAS..10015387W. doi:10.1073/pnas.2536810100. PMC 307577. PMID 14657349. https://www.ncbi.nlm.nih.gov/pmc/articles/PMC307577/.
^ “Adenosine nucleotide modulates the physical interaction between hMSH2 and BRCA1”. Oncogene 20 (34): 4640?9. (August 2001). doi:10.1038/sj.onc.1204625. PMID 11498787.
^ a b “BASC, a super complex of BRCA1-associated proteins involved in the recognition and repair of aberrant DNA structures”. Genes Dev. 14 (8): 927?39. (April 2000). doi:10.1101/gad.14.8.927. PMC 316544. PMID 10783165. https://www.ncbi.nlm.nih.gov/pmc/articles/PMC316544/.
^ “Methylator-induced, mismatch repair-dependent G2 arrest is activated through Chk1 and Chk2”. Mol. Biol. Cell 16 (3): 1513?26. (March 2005). doi:10.1091/mbc.E04-02-0089. PMC 551512. PMID 15647386. https://www.ncbi.nlm.nih.gov/pmc/articles/PMC551512/.
^ “The mismatch repair system is required for S-phase checkpoint activation”. Nat. Genet. 33 (1): 80?4. (January 2003). doi:10.1038/ng1052. PMID 12447371.

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出典: フリー百科事典『ウィキペディア（Wikipedia）』
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