興奮毒性 - 暇つぶしWikipedia

興奮毒性

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^ Kim AH, Kerchner GA, and Choi DW. Blocking Excitotoxicity or Glutamatergic Storm. Chapter 1 in CNS Neuroprotection. Marcoux FW and Choi DW, editors. Springer, New York. 2002. Pages 3-36
^ Hughes JR (February 2009). “Alcohol withdrawal seizures”. Epilepsy Behav 15 (2): 92?7. doi:10.1016/j.yebeh.2009.02.037. PMID 19249388.
^ Camacho, A; Massieu, L (2006). “Role of glutamate transporters in the clearance and release of glutamate during ischemia and its relation to neuronal death.”. Archives of Medical Research 37 (1): 11?8. doi:10.1016/j.arcmed.2005.05.014. PMID 16314180.
^ Fujikawa, DG (2005). “Prolonged seizures and cellular injury: understanding the connection.”. Epilepsy & Behavior 7 Suppl 3: S3?11. doi:10.1016/j.yebeh.2005.08.003. PMID 16278099.
^ Watkins, Jeffrey C; Jane, David E (2 February 2009). “The glutamate story”. British Journal of Pharmacology 147 (S1): S100?S108. doi:10.1038/sj.bjp.0706444. PMC 1760733. PMID 16402093. https://www.ncbi.nlm.nih.gov/pmc/articles/PMC1760733/.
^ Lucas, DR; Newhouse, JP (1957). “The toxic effect of sodium L-glutamate on the inner layers of the retina.”. Archives of Ophthalmology 58 (2): 193?201. doi:10.1001/archopht.1957.00940010205006. PMID 13443577.
^ Olney, JW (1969). “Brain lesions, obesity, and other disturbances in mice treated with monosodium glutamate.”. Science 164 (3880): 719?21. Bibcode: 1969Sci...164..719O. doi:10.1126/science.164.3880.719. hdl:10217/207298. PMID 5778021.
^ Hardingham, G. E.; Fukunaga, Y.; Bading, H. (May 2002). “Extrasynaptic NMDARs oppose synaptic NMDARs by triggering CREB shut-off and cell death pathways”. Nature Neuroscience 5 (5): 405?414. doi:10.1038/nn835. ISSN 1097-6256. PMID 11953750. https://pubmed.ncbi.nlm.nih.gov/11953750.
^ Yan, Jing; Bengtson, C. Peter; Buchthal, Bettina; Hagenston, Anna M.; Bading, Hilmar (9 October 2020). “Coupling of NMDA receptors and TRPM4 guides discovery of unconventional neuroprotectants”. Science 370 (6513). doi:10.1126/science.aay3302. ISSN 1095-9203. PMID 33033186. https://pubmed.ncbi.nlm.nih.gov/33033186.
^ Head trauma : basic, preclinical, and clinical directions. Leonard P. Miller, Ronald L. Hayes, Jennifer K. Newcomb. New York: Wiley-Liss. (2001). pp. 87-113. ISBN 0-471-36015-5. OCLC 45618924. https://www.worldcat.org/oclc/45618924
^ Clements, JD; Lester, RA; Tong, G; Jahr, CE; Westbrook, GL (1992). “The time course of glutamate in the synaptic cleft”. Science 258 (5087): 1498?501. Bibcode: 1992Sci...258.1498C. doi:10.1126/science.1359647. PMID 1359647.
^ Yang Derek D. (October 1997). “Absence of excitotoxicity-induced apoptosis in the hippocampus of mice lacking the Jnk3 gene”. Nature 389 (6653): 865?870. Bibcode: 1997Natur.389..865Y. doi:10.1038/39899. PMID 9349820.
^ Ankarcrona Maria (October 1995). “Glutamate-induced neuronal death: A succession of necrosis or apoptosis depending on mitochondrial function”. Neuron 15 (4): 961?973. doi:10.1016/0896-6273(95)90186-8. PMID 7576644.
^ Hulsebosch (Apr 2009). “Mechanisms of chronic central neuropathic pain after spinal cord injury”. Brain Res Rev 60 (1): 202?13. doi:10.1016/j.brainresrev.2008.12.010. PMC 2796975. PMID 19154757. https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2796975/.
^ Nakamura (Aug 2010). “S-nitrosylation of Drp1 links excessive mitochondrial fission to neuronal injury in neurodegeneration”. Mitochondrion 10 (5): 573?8. doi:10.1016/j.mito.2010.04.007. PMC 2918703. PMID 20447471. https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2918703/.
^ a b Dutta (Jan 2011). “Mechanisms of neuronal dysfunction and degeneration in multiple sclerosis”. Prog Neurobiol 93 (1): 1?12. doi:10.1016/j.pneurobio.2010.09.005. PMC 3030928. PMID 20946934. https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3030928/.
^ Stavrovskaya, IG; Kristal, BS (2005). “The powerhouse takes control of the cell: is the mitochondrial permeability transition a viable therapeutic target against neuronal dysfunction and death?”. Free Radical Biology & Medicine 38 (6): 687?97. doi:10.1016/j.freeradbiomed.2004.11.032. PMID 15721979.
^ Allen, J; Romay-Tallon, R; Brymer, K; Caruncho, H; Kalynchuk, L (2018). “Mitochondria and Mood: Mitochondrial Dysfunction as a Key Player in the Manifestation of Depression”. Frontiers in Neuroscience 12: 386. doi:10.3389/fnins.2018.00386. PMC 5997778. PMID 29928190. https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5997778/.
^ Li (2001). “Na+-K+-ATPase inhibition and depolarization induce glutamate release via reverse Na+-dependent transport in spinal cord white matter”. Neuroscience 107 (4): 675?683. doi:10.1016/s0306-4522(01)00385-2. PMID 11720790.
^ Basic neurochemistry : molecular, cellular, and medical aspects. George J. Siegel (6th ed ed.). Philadelphia: Lippincott Williams & Wilkins. (1999). ISBN 0-397-51820-X. OCLC 39013748. https://www.worldcat.org/oclc/39013748
^ a b Hardingham, GE; Fukunaga, Y; Bading, H (2002). “Extrasynaptic NMDARs oppose synaptic NMDARs by triggering CREB shut-off and cell death pathways”. Nature Neuroscience 5 (5): 405?14. doi:10.1038/nn835. PMID 11953750.
^ Hardingham, Giles E.; Bading, Hilmar (2010). “Synaptic versus extrasynaptic NMDA receptor signalling: implications for neurodegenerative disorders”. Nature Reviews Neuroscience 11 (10): 682?696. doi:10.1038/nrn2911. PMC 2948541. PMID 20842175. https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2948541/.
^ Brand, LE (2009). “Human exposure to cyanobacteria and BMAA”. Amyotrophic Lateral Sclerosis 20: 85?95. doi:10.3109/17482960903273585. PMID 19929739.
^ Vyas, KJ; Weiss, JH (2009). “BMAA--an unusual cyanobacterial neurotoxin”. Amyotrophic Lateral Sclerosis 10: 50?55. doi:10.3109/17482960903268742. PMID 19929732.
^ a b Chiu, AS (2012). “Excitotoxic potential of the cyanotoxin β-methyl-amino-l-alanine (BMAA) in primary human neurons”. Toxicon 60 (6): 1159?1165. doi:10.1016/j.toxicon.2012.07.169. PMID 22885173.
^ Papapetropolous, S (2007). “Is there a role for naturally occurring cyanobacterial toxins in neurodegeneration? The beta-N-methylamino-L-alanine (BMAA) paradigm.”. Neurochemistry International 50 (7): 998?1003. doi:10.1016/j.neuint.2006.12.011. PMID 17296249.

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