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^ “The multifaceted cardiac sodium channel and its clinical implications”. Heart 98 (17): 1318?24. (September 2012). doi:10.1136/heartjnl-2012-301784. PMID 22875823.
^ “Loss-of-function of the voltage-gated sodium channel NaV1.5 (channelopathies) in patients with irritable bowel syndrome”. Gastroenterology 146 (7): 1659?1668. (June 2014). doi:10.1053/j.gastro.2014.02.054. PMC 4096335. PMID 24613995. https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4096335/.
^ “A SCN9A gene-encoded dorsal root ganglia sodium channel polymorphism associated with severe fibromyalgia”. BMC Musculoskeletal Disorders 13: 23. (February 2012). doi:10.1186/1471-2474-13-23. PMC 3310736. PMID 22348792. https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3310736/.
^ “From ionic currents to molecular mechanisms: the structure and function of voltage-gated sodium channels”. Neuron 26 (1): 13?25. (April 2000). doi:10.1016/S0896-6273(00)81133-2. PMID 10798388.
^ “Primary structure and functional expression of the beta 1 subunit of the rat brain sodium channel”. Science 256 (5058): 839?42. (May 1992). Bibcode: 1992Sci...256..839I. doi:10.1126/science.1375395. PMID 1375395.
^ “Sodium channel beta subunits mediate homophilic cell adhesion and recruit ankyrin to points of cell-cell contact”. The Journal of Biological Chemistry 275 (15): 11383?8. (April 2000). doi:10.1074/jbc.275.15.11383. PMID 10753953.
^ “Structural requirements for interaction of sodium channel beta 1 subunits with ankyrin”. The Journal of Biological Chemistry 277 (29): 26681?8. (July 2002). doi:10.1074/jbc.M202354200. PMID 11997395.
^ “Contactin associates with Na+ channels and increases their functional expression”. The Journal of Neuroscience 21 (19): 7517?25. (October 2001). doi:10.1523/JNEUROSCI.21-19-07517.2001. PMID 11567041.
^ “Interaction of voltage-gated sodium channels with the extracellular matrix molecules tenascin-C and tenascin-R”. Proceedings of the National Academy of Sciences of the United States of America 95 (26): 15753?7. (December 1998). Bibcode: 1998PNAS...9515753S. doi:10.1073/pnas.95.26.15753. PMC 28116. PMID 9861042. https://www.ncbi.nlm.nih.gov/pmc/articles/PMC28116/.
^ “Sodium channel beta1 and beta3 subunits associate with neurofascin through their extracellular immunoglobulin-like domain”. The Journal of Cell Biology 154 (2): 427?34. (July 2001). doi:10.1083/jcb.200102086. PMC 2150779. PMID 11470829. https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2150779/.
^ a b c “Brugada Syndrome: Clinical, Genetic, Molecular, Cellular, and Ionic Aspects”. Current Problems in Cardiology 41 (1): 7?57. (January 2016). doi:10.1016/j.cpcardiol.2015.06.002. PMC 4737702. PMID 26671757. https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4737702/.
^ “Electrophysiological analysis of the neurotoxic action of a funnel-web spider toxin, delta-atracotoxin-HV1a, on insect voltage-gated Na+ channels”. The Journal of Experimental Biology 204 (Pt 4): 711?21. (February 2001). PMID 11171353.
^ “Scorpion toxins specific for Na+-channels”. European Journal of Biochemistry 264 (2): 287?300. (September 1999). doi:10.1046/j.1432-1327.1999.00625.x. PMID 10491073.
^ “pH Modulation of Voltage-Gated Sodium Channels”. Voltage-gated Sodium Channels: Structure, Function and Channelopathies. Handbook of Experimental Pharmacology. 246. (February 2018). pp. 147?160. doi:10.1007/164_2018_99. ISBN 978-3-319-90283-8. PMID 29460150
^ “Effects of external protons on single cardiac sodium channels from guinea pig ventricular myocytes”. The Journal of General Physiology 98 (6): 1065?83. (December 1991). doi:10.1085/jgp.98.6.1065. PMC 2229074. PMID 1664454. https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2229074/.
^ a b “On the structural basis for size-selective permeation of organic cations through the voltage-gated sodium channel. Effect of alanine mutations at the DEKA locus on selectivity, inhibition by Ca2+ and H+, and molecular sieving”. The Journal of General Physiology 110 (6): 693?715. (December 1997). doi:10.1085/jgp.110.6.693. PMC 2229404. PMID 9382897. https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2229404/.
^ “Role of outer ring carboxylates of the rat skeletal muscle sodium channel pore in proton block”. The Journal of Physiology 543 (Pt 1): 71?84. (August 2002). doi:10.1113/jphysiol.2002.021014. PMC 2290475. PMID 12181282. https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2290475/.
^ “Acidosis differentially modulates inactivation in na(v)1.2, na(v)1.4, and na(v)1.5 channels”. Frontiers in Pharmacology 3: 109. (2012). doi:10.3389/fphar.2012.00109. PMC 3372088. PMID 22701426. https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3372088/.
^ “Proton sensors in the pore domain of the cardiac voltage-gated sodium channel”. The Journal of Biological Chemistry 288 (7): 4782?91. (February 2013). doi:10.1074/jbc.M112.434266. PMC 3576083. PMID 23283979. https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3576083/.
^ “Isoform-dependent interaction of voltage-gated sodium channels with protons”. The Journal of Physiology 576 (Pt 2): 493?501. (October 2006). doi:10.1113/jphysiol.2006.115659. PMC 1890365. PMID 16873405. https://www.ncbi.nlm.nih.gov/pmc/articles/PMC1890365/.
^ “Blood and muscle pH after maximal exercise in man”. Journal of Applied Physiology 32 (3): 304?8. (March 1972). doi:10.1152/jappl.1972.32.3.304. PMID 5010039.
^ “A Mixed Periodic Paralysis & Myotonia Mutant, P1158S, Imparts pH-Sensitivity in Skeletal Muscle Voltage-gated Sodium Channels”. Scientific Reports 8 (1): 6304. (April 2018). Bibcode: 2018NatSR...8.6304G. doi:10.1038/s41598-018-24719-y. PMC 5908869. PMID 29674667. https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5908869/.
^ “Effects of acidosis on neuronal voltage-gated sodium channels: Nav1.1 and Nav1.3”. Channels 12 (1): 367?377. (October 2018). doi:10.1080/19336950.2018.1539611. PMC 6284583. PMID 30362397. https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6284583/.
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